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However, many interesting metabolic enzymes, redox-sensing transcription factors or virulence factors might be controlled by protein S -thiolations in the pathogenic bacteria S

GSH production is supported by NRF2 through controlling several enzymes responsible for GSH synthesis, such as the glutamate-cysteine ligase (GCL) complex composed of two subunits, the modifier (GCLM) subunit and the catalytic (GCLC) subunit, which catalyze the rate-limiting step of GSH synthesis that converts cysteine and glutamate into GSH [22]

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Kulbay M, et al

ML, 0.5 mm

Furthermore, it is crucial to consider the investigation of subclinical alterations, whether genetic or metabolic, that may necessitate higher thiamine levels than what current testing parameters indicate [7]